40 HZ SOUND WAVE EFFECTS ON CARDIOVASCULAR SYSTEM: NITRIC OXIDE RELEASE IN COLLEGE-AGED INDIVIDUALS
Department
Applied Health Sciences
Advisor
Christopher Carroll
Document Type
Event
Version
Metadata Only
Abstract
Research is limited on the physical effects of sound waves on the human body. This study investigates whether sound waves can stimulate endothelial cells to release Nitric oxide (NO), potentially impacting cardiovascular metrics like blood pressure (BP), blood oxygenation (O2), and heart rate (HR), without involving the auditory response. Methods: Twenty-four Bethel University students (14 males, 10 females, average age 21.67 ± 1.34 years) were randomly assigned to one of two groups: sound (S) or no sound (NS). They attended two sessions within 10 days. During each session, BP was measured before and after 15 minutes of sound exposure or no sound. HR and O2 were recorded before, during, and immediately after each 15-minute session. The paired samples t-test on SPSS V28 showed no significant change in BP, HR, or O2 in the S and NS groups after 40 Hz exposure. However, comparing between groups, those exposed to sound had significantly higher O2 (p = 0.038) and lower HR (p = 0.005). With no significant changes in BP with sound exposure, the increase in O2 and decrease in HR suggest NO directly modulating HR independent of baroreflex modulation.
Recommended Citation
Doherty, Ben; Johnson, Lucas; and Paradis, Seth, "40 HZ SOUND WAVE EFFECTS ON CARDIOVASCULAR SYSTEM: NITRIC OXIDE RELEASE IN COLLEGE-AGED INDIVIDUALS" (2024). Science Symposium. 4.
https://spark.bethel.edu/science_symposium/spring2024/schedule2024/4
40 HZ SOUND WAVE EFFECTS ON CARDIOVASCULAR SYSTEM: NITRIC OXIDE RELEASE IN COLLEGE-AGED INDIVIDUALS
Research is limited on the physical effects of sound waves on the human body. This study investigates whether sound waves can stimulate endothelial cells to release Nitric oxide (NO), potentially impacting cardiovascular metrics like blood pressure (BP), blood oxygenation (O2), and heart rate (HR), without involving the auditory response. Methods: Twenty-four Bethel University students (14 males, 10 females, average age 21.67 ± 1.34 years) were randomly assigned to one of two groups: sound (S) or no sound (NS). They attended two sessions within 10 days. During each session, BP was measured before and after 15 minutes of sound exposure or no sound. HR and O2 were recorded before, during, and immediately after each 15-minute session. The paired samples t-test on SPSS V28 showed no significant change in BP, HR, or O2 in the S and NS groups after 40 Hz exposure. However, comparing between groups, those exposed to sound had significantly higher O2 (p = 0.038) and lower HR (p = 0.005). With no significant changes in BP with sound exposure, the increase in O2 and decrease in HR suggest NO directly modulating HR independent of baroreflex modulation.